DTIC ADA228679: Effect of Prostaglandin E in pdf

DTIC ADA228679: Effect of Prostaglandin E in

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One of the primary aetiologies for mortality following burn injury is the development of infectious complications (Sevitt, 1979). These infections have two main causes, a loss of the skin’s natural barrier and the immunosuppression that results from burn injuries. This immunosuppression is due to a number of factors, including inadequate nutrition in the postburn period, the use of immunosuppressive agents such as anaesthesia and blood transfusions, and the release of endogenous immunosuppressive metabolites. Prostaglandin E (PGE) has been reported to be one of the immunosuppressive metabolites released following burn injury (Ninnemann and Stockland, 1984). The belief in an immunosuppressive nature of PGE has resulted from two areas of investigation. (js

• Creator/s: Defense Technical Information Center
• Date: 1/1/1990
• Year: 1990
• Book Topics/Themes: DTIC Archive, Waymack, J P, ARMY INST OF SURGICAL RESEARCH FORT SAMHOUSTON TX, *BURNS(INJURIES), MODELS, EXPERIMENTAL DESIGN, RESISTANCE, BARRIERS, INFECTIOUS DISEASES, NUTRITION, BLOOD, METABOLITES, IMMUNOSUPPRESSION, BLOOD TRANSFUSION, SEPSIS, PROSTAGLANDIN

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